Our strategy is to 1) think through the effectiveness of 100mg daily enteric coated Aspirin and do a little novelty to hve a gut ecossytem that might manage the mucin releasing cells and 2) look at the gene deletions for dna repair and see if we can find a workaround.
background resource files
question 1 : can we part replace aspirin with low poisonous salicyalte rich foods low in glyphosate?
as poison
in foods and action
Plant hormone
Salicylic acid (SA) is a phenolic phytohormone and is found in plants with roles in plant growth and development, photosynthesis, transpiration, ion uptake and transport. SA also induces specific changes in leaf anatomy and chloroplast structure. SA is involved in endogenous signaling, mediating in plant defense against pathogens.[4] It plays a role in the resistance to pathogens by inducing the production of pathogenesis-related proteins.[5] It is involved in the systemic acquired resistance (SAR) in which a pathogenic attack on one part of the plant induces resistance in other parts. The signal can also move to nearby plants by salicylic acid being converted to the volatile ester, methyl salicylate.[6]Mechanism of action
Salicylic acid has been shown to work through several different pathways. It produces its anti-inflammatory effects via suppressing the activity of cyclooxygenase (COX), an enzyme that is responsible for the production of pro-inflammatory mediators such as the prostaglandins. It does this not by direct inhibition of COX like most other non-steroidal anti-inflammatory drugs (NSAIDs) but instead by suppression of the expression of the enzyme (via a yet-unelucidated mechanism).[22] Salicylic acid has also been shown to activate adenosine monophosphate-activated protein kinase (AMPK), and it is thought that this action may play a role in the anticancer effects of the compound and its prodrugs aspirin and salsalate. In addition, the antidiabetic effects of salicylic acid are likely mediated by AMPK activation primarily through allosteric conformational change that increases levels of phosphorylation.[23] Salicylic acid also uncouples oxidative phosphorylation, which leads to increased ADP:ATP and AMP:ATP ratios in the cell. As a consequence, salicylic acid may alter AMPK activity and subsequently exert its anti-diabetic properties through altered energy status of the cell. Even in AMPK knock-out mice, however, there is an anti-diabetic effect, demonstrating that there is at least one additional, yet-unidentified action of the compound.[24]Bacillis as an Agent
Grdening Australia link
Christal site for Lucy
Is something interfering with Trypophan uptake?
Cancer statisitcs in Sri Lanka by Relgion: eat like a moslem female
fungus to control other fungus as ecosystem
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